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Aaron Hernandez's Brain


Paa Langfart

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7 minutes ago, CPcavedweller said:

Humans contribute less than 3% of total carbon output on Earth. Do humans contribute to the warming? It's possible but it's not exactly measurable. One can assume but assumptions that haven't been or can't be proven aren't science. Not only that, but as carbon causes a warming effect, ice melts, effectively speeding up the warming process because insulators are being removed while carbon is still increasing. Eventually, this will disrupt the ocean currents due to water temperature and density differences, leading to a lack of re-distribution of wealth warmth, causing the Poles to freeze again and effectively causing the next ice age.

This isn't from a movie. It's real life and it has happened previously. We are in the middle of an interglacial period, an interglacial period that scientists say is warmer than it should be at this point. But meteorologists can't predict weather correctly 5 days out, so why would a climatologist be able to predict the climate 10,000 years out.

Last point, scientists argue that it is the warmest it's been in the 10's of thousands of years. Earth is 4,600,000,000 years old. So you have a comparison of 10,000 to 4,600,000,000. Its statistically insignificant and irrelevant to assume that science is infallible. 

And no, the Earth isn't flat and there is no conspiracy with Vegas. Though I do think the death of a DNC Staffer in Washington D.C. in a supposed robbery gone wrong is suspicious. B613. 

agree with all your points except the bolded. when you have people who survived the attack only to magically end up dead a few weeks later definitely raises some suspicion. google Dennis and Lorraine Carver and Kymberley Suchomel

so far that's 3, one of whom went on record to say she saw multiple shooters including a shooter on ground level. This is not un common at all. There were many survivors of 9/11 who mysteriously ended up dead, there's been doctors who've introduced potential cures for cancer who were killed. Of course, can't leave out David Crawly, the creator of Gray State which exposed the inevitable martial law and FEMA camps in the US, who was found dead along with his wife, and kid in an apparent murder suicide. And like you pointed out, the death of Seth Rich who exposed Hilary's emails to Assange and wikileaks. 

look, i try not to be a conspiracy nut, but when facts and common sense tell you otherwise, it's hard to believe the official narratives and anything the mainstream media puts out there. 

 

 

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12 minutes ago, RealisticPanther said:

No...I have a biology and chemistry degree, I'm science-minded, I believe in truth, science, and objectivity. I know how to interpret studies and results properly. Anyone science minded with logic who looks at CTE and the evidence associated with it would conclude the same thing as I just have...that its a bunch of BS. Marijuana users have drastically different looking brains than non-users, too, but there isn't a significant cognitive decline associated with the brain changes. This is the same with CTE...just because the brain looks different due to adaptation doesn't mean there's some huge cognitive decline or severe pathology going on, especially in the absence of any evidence at all as is the case with CTE.

 

9 minutes ago, RealisticPanther said:

Now if you believe CTE causes significant cognitive impairment or negative effects and possibly leads to increased occurrence of long term pathology, then you also have to admit marijuana does, because marijuana has monumentally more evidence of causing cognitive decline than CTE does..because CTE has zero. absolutely none. 

Theres plenty of evidence. Are you just being intentionally obtuse? Theres tons of peer reviewed studies on the effects of TBI including CTE and the affect they have on all sorts of brain functions.

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16 minutes ago, Floppin said:

 

Theres plenty of evidence. Are you just being intentionally obtuse? Theres tons of peer reviewed studies on the effects of TBI including CTE and the affect they have on all sorts of brain functions.

no, there isn't, i'm not going to argue with you. TBI doesn't equal CTE. If you want to argue, argue with google scholar and pubmed. If you can cite me one article showing what you just stated ill suck my own cock.

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15 minutes ago, E CaT PanTHer 2 said:

agree with all your points except the bolded. when you have people who survived the attack only to magically end up dead a few weeks later definitely raises some suspicion. google Dennis and Lorraine Carver and Kymberley Suchomel

so far that's 3, one of whom went on record to say she saw multiple shooters including a shooter on ground level. This is not un common at all. There were many survivors of 9/11 who mysteriously ended up dead, there's been doctors who've introduced potential cures for cancer who were killed. Of course, can't leave out David Crawly, the creator of Gray State which exposed the inevitable martial law and FEMA camps in the US, who was found dead along with his wife, and kid in an apparent murder suicide. And like you pointed out, the death of Seth Rich who exposed Hilary's emails to Assange and wikileaks. 

look, i try not to be a conspiracy nut, but when facts and common sense tell you otherwise, it's hard to believe the official narratives and anything the mainstream media puts out there. 

 

 

The government elite is nothing to fug with. I've researched this topic for a long time now, and let me just say it is very scary and very real.

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10 minutes ago, RealisticPanther said:

no, there isn't, i'm not going to argue with you. TBI doesn't equal CTE. If you want to argue, argue with google scholar and pubmed. If you can cite me one article showing what you just stated ill suck my own cock.

Abstract

Traumatic brain injury (TBI) results in significant disability due to cognitive deficits particularly in attention, learning and memory, and higher-order executive functions. The role of TBI in chronic neurodegeneration and the development of neurodegenerative diseases including Alzheimer's disease (AD), Parkinson's disease (PD), Amyotrophic Lateral Sclerosis (ALS) and most recently chronic traumatic encephalopathy (CTE) is of particular importance. However, despite significant effort very few therapeutic options exist to prevent or reverse cognitive impairment following TBI. In this review, we present experimental evidence of the known secondary injury mechanisms which contribute to neuronal cell loss, axonal injury, and synaptic dysfunction and hence cognitive impairment both acutely and chronically following TBI. In particular we focus on the mechanisms linking TBI to the development of two forms of dementia: AD and CTE. We provide evidence of potential molecular mechanisms involved in modulating Aβ and Tau following TBI and provide evidence of the role of these mechanisms in AD pathology. Additionally we propose a mechanism by which Aβ generated as a direct result of TBI is capable of exacerbating secondary injury mechanisms thereby establishing a neurotoxic cascade that leads to chronic neurodegeneration

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3705200/

No TBI =/= CTE but it is one disorder of many that has been shown to develop due to the affect of various types of TBI. The above link goes into this and how they (including CTE) affect cognition. There's many more but thats a start for you

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11 minutes ago, Floppin said:

Abstract

Traumatic brain injury (TBI) results in significant disability due to cognitive deficits particularly in attention, learning and memory, and higher-order executive functions. The role of TBI in chronic neurodegeneration and the development of neurodegenerative diseases including Alzheimer's disease (AD), Parkinson's disease (PD), Amyotrophic Lateral Sclerosis (ALS) and most recently chronic traumatic encephalopathy (CTE) is of particular importance. However, despite significant effort very few therapeutic options exist to prevent or reverse cognitive impairment following TBI. In this review, we present experimental evidence of the known secondary injury mechanisms which contribute to neuronal cell loss, axonal injury, and synaptic dysfunction and hence cognitive impairment both acutely and chronically following TBI. In particular we focus on the mechanisms linking TBI to the development of two forms of dementia: AD and CTE. We provide evidence of potential molecular mechanisms involved in modulating Aβ and Tau following TBI and provide evidence of the role of these mechanisms in AD pathology. Additionally we propose a mechanism by which Aβ generated as a direct result of TBI is capable of exacerbating secondary injury mechanisms thereby establishing a neurotoxic cascade that leads to chronic neurodegeneration

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3705200/

No TBI =/= CTE but it is one disorder of many that has been shown to develop due to the affect of various types of TBI. The above link goes into this and how they (including CTE) affect cognition. There's many more but thats a start for you

I'm not even going to begin to get in how flawed what you just cited me was, right now at least, but let's start with how the authors say "recently its become apparent (mTBI's that may or may not be caused by contact sports) can lead to significant emotional and cognitive disabilities" and then they cite a study to support that statement which is nothing but conjecture and doesn't prove there are long term (a term subjective in itself) consequences at all. 

There's absolutely no concrete data or epidemiology to suggest CTE causes an increased incidence of neurological disease or long term cognitive impairment. We can logically speculate that it could cause some cognitive problems, however, they appear to be insignificant and very mild if they exist. People with depression, ptsd...their brains look the same as someone with CTE. But i'm sure i'll come back and list the rest of the flaws later.

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2 hours ago, CPcavedweller said:

Humans contribute less than 3% of total carbon output on Earth. Do humans contribute to the warming? It's possible but it's not exactly measurable. One can assume but assumptions that haven't been or can't be proven aren't science. Not only that, but as carbon causes a warming effect, ice melts, effectively speeding up the warming process because insulators are being removed while carbon is still increasing. Eventually, this will disrupt the ocean currents due to water temperature and density differences, leading to a lack of re-distribution of wealth warmth, causing the Poles to freeze again and effectively causing the next ice age.

This isn't from a movie. It's real life and it has happened previously. We are in the middle of an interglacial period, an interglacial period that scientists say is warmer than it should be at this point. But meteorologists can't predict weather correctly 5 days out, so why would a climatologist be able to predict the climate 10,000 years out.

Last point, scientists argue that it is the warmest it's been in the 10's of thousands of years. Earth is 4,600,000,000 years old. So you have a comparison of 10,000 to 4,600,000,000. Its statistically insignificant and irrelevant to assume that science is infallible. 

And no, the Earth isn't flat and there is no conspiracy with Vegas. Though I do think the death of a DNC Staffer in Washington D.C. in a supposed robbery gone wrong is suspicious. B613. 

Nailed it

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2 hours ago, Floppin said:

Abstract

Traumatic brain injury (TBI) results in significant disability due to cognitive deficits particularly in attention, learning and memory, and higher-order executive functions. The role of TBI in chronic neurodegeneration and the development of neurodegenerative diseases including Alzheimer's disease (AD), Parkinson's disease (PD), Amyotrophic Lateral Sclerosis (ALS) and most recently chronic traumatic encephalopathy (CTE) is of particular importance. However, despite significant effort very few therapeutic options exist to prevent or reverse cognitive impairment following TBI. In this review, we present experimental evidence of the known secondary injury mechanisms which contribute to neuronal cell loss, axonal injury, and synaptic dysfunction and hence cognitive impairment both acutely and chronically following TBI. In particular we focus on the mechanisms linking TBI to the development of two forms of dementia: AD and CTE. We provide evidence of potential molecular mechanisms involved in modulating Aβ and Tau following TBI and provide evidence of the role of these mechanisms in AD pathology. Additionally we propose a mechanism by which Aβ generated as a direct result of TBI is capable of exacerbating secondary injury mechanisms thereby establishing a neurotoxic cascade that leads to chronic neurodegeneration

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3705200/

No TBI =/= CTE but it is one disorder of many that has been shown to develop due to the affect of various types of TBI. The above link goes into this and how they (including CTE) affect cognition. There's many more but thats a start for you

yea bro, i read that study, its complete garbage, all the studies they cite are poorly done, inconclusive, or simply conjecture. They give a bunch of science of how things work which sounds good to people who don't know how to interpret studies and go "hmmm, axonal damage and transduction pathway names and big words, sounds legit"...but its all a bunch of filler. Yes, that's how any damage, long term, short term, from brief inflammation, whatever works...and? The studies they cite as "proof" are as I said simply conjecture...or purposely misinterpreted. The studies they cite showing that football players have "3x the instance of mortality from neurological pathology" ...well football players have half of the overall mortality of the general population...they live a lot longer than the normal population so no shyt the risk of dying from neurological diseases is gonna increase when you live longer.

I hope that if you feel the need to have an opinion about this you reconsider your position or how strongly you're convinced of your position, because, to put it bluntly, you're wrong and obviously don't have the deductive reasoning skills or know how to interpret the studies you get your information from to distinguish good information and conclusions from bad. 

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